Svetlana B. Poplin v. Amerisure Insurance Company ( 2013 )


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  • Opinion issued October 31, 2013
    In The
    Court of Appeals
    For The
    First District of Texas
    ————————————
    NO. 01-13-00102-CV
    ———————————
    SVETLANA B. POPLIN, Appellant
    V.
    AMERISURE INSURANCE COMPANY, Appellee
    On Appeal from the County Civil Court at Law No 2
    Harris County, Texas
    Trial Court Case No. 907501
    MEMORANDUM OPINION
    This is a worker’s compensation case. Appellant Svetlana Poplin appeals
    from the trial court’s order granting summary judgment in Appellee Amerisure
    Insurance Company’s favor. We affirm.
    BACKGROUND
    Poplin’s husband, James Poplin, worked as an air conditioning technician
    for Air Performance Service, Inc. Poplin’s petition alleges that, on June 25, 2006,
    James worked on an air-conditioning tower located atop of a six-story building.
    After a few hours, he became ill. Paramedics called to the scene diagnosed James
    with heat exhaustion and transported him to the hospital while infusing saline to
    replace lost fluids.   Within twenty minutes of reaching the hospital, James’s
    condition was downgraded to cardiac arrest and he passed away shortly thereafter.
    A. Administrative Proceedings
    Poplin applied for workers’ compensation benefits from Amerisure, Air
    Performance’s provider.    Amerisure denied her claim.      After an unsuccessful
    mediation, a Benefit Contested Case Hearing was held to “resolve the following
    disputed issue: Whether James Poplin sustained a compensable fatal heart attack
    on June 25, 2006.”     Both parties were represented by counsel and presented
    evidence.   The hearing officer’s report noted that Poplin “presented several
    medical articles to support the proposition that physical stress could constitute a
    cause of a heart attack,” but “she offered no medical evidence specific to this case
    to indicate to what extent Mr. Poplin’s work activities of June 25, 2006 caused or
    contributed to his heart attack.” Amerisure “offered the opinions of Drs. Chu and
    Podet, who indicated that [James’] work, rather than the natural [progression] of
    2
    his preexisting heart disease, was not a substantial contributing factor in his heart
    attack.” The report contained several express findings, including:
    -“James Poplin’s heart attack on June 25, 2006 occurred at a definite
    time and place.”
    -“James Poplin’s heart attack of June 25, 2006 was not triggered
    solely by emotional stress.”
    -“James Poplin’s heart attack of June 25, 2006 was not caused by a
    specific event occurring within the course and scope of his
    employment.”
    -“James Poplin’s work, rather than the natural progression of his
    preexisting heart condition or disease, was not a substantial
    contributing factor of his heart attack of June 25, 2006.”
    The report concluded that James “did not sustain a compensable fatal heart
    attack.” Poplin’s claim was accordingly denied. This decision was affirmed by the
    Division of Workers’ Compensation Appeals Panel.
    B. Trial Court Proceedings
    In February 2008, Poplin filed suit in county court, seeking review of the
    denial of benefits.1 As the party appealing the final administrative decision on the
    compensability of an injury, she bore “the burden of proof by a preponderance of
    the evidence.” TEX. LABOR CODE ANN § 410.303 (Vernon 2006); Morales v.
    Liberty Mut. Ins. Co., 
    241 S.W.3d 514
    , 516 (Tex. 2007). In July 2012, following
    1
    “A party that has exhausted its administrative remedies . . . and that is aggrieved
    by a final decision . . . may seek judicial review.” TEX. LAB. CODE § 410.251
    (Vernon 2006).
    3
    expiration of the discovery period, Amerisure moved for traditional and no-
    evidence summary judgment.        The trial court granted summary judgment in
    Amerisure’s favor without specifying the grounds. It is from that order that Poplin
    now appeals.
    STANDARD OF REVIEW
    An appellate court reviews de novo the trial court’s ruling on a summary
    judgment motion. Mann Frankfort Stein & Lipp Advisors, Inc. v. Fielding, 
    289 S.W.3d 844
    , 848 (Tex. 2009); Valence Operating Co. v. Dorsett, 
    164 S.W.3d 656
    ,
    661 (Tex. 2005). When the trial court does not specify the grounds for its grant of
    summary judgment, the reviewing court must affirm the summary judgment if any
    of the theories presented to the court and preserved for appeal are meritorious. See
    Provident Life & Accident Ins. Co. v. Knott, 
    128 S.W.3d 211
    , 216 (Tex. 2003).
    When there are multiple grounds for summary judgment and the order does not
    specify the ground on which the summary judgment was granted, the appellant
    must negate all grounds on appeal. State Farm Fire & Cas. Co. v. S.S., 
    858 S.W.2d 374
    , 381 (Tex. 1993); Ellis v. Precision Engine Rebuilders, Inc., 
    68 S.W.3d 894
    ,
    898 (Tex. App.—Houston [1st Dist.] 2002, no pet.).
    Traditional summary judgment is proper only when the movant establishes
    that there is no genuine issue of material fact and that the movant is entitled to
    judgment as a matter of law. TEX. R. CIV. P. 166a(c). In reviewing a traditional
    4
    summary judgment, we must indulge every reasonable inference in favor of the
    nonmovant, take all evidence favorable to the nonmovant as true, and resolve any
    doubts in favor of the nonmovant. Texas Commerce Bank v. Grizzle, 
    96 S.W.3d 240
    , 252 (Tex. 2002). A defendant who moves for traditional summary judgment
    on the plaintiff’s claims must conclusively disprove at least one element of each of
    the plaintiff’s causes of action. Little v. Tex. Dep’t of Criminal Justice, 
    148 S.W.3d 374
    , 381 (Tex. 2004).
    A no-evidence motion for summary judgment is essentially a directed
    verdict granted before trial, to which we apply a legal sufficiency standard of
    review. King Ranch, Inc. v. Chapman, 
    118 S.W.3d 742
    , 750–51 (Tex. 2003). In
    general, a party seeking a no-evidence summary judgment must assert that no
    evidence exists as to one or more of the essential elements of the nonmovant’s
    claims on which it would have the burden of proof at trial. Holmstrom v. Lee, 
    26 S.W.3d 526
    , 530 (Tex. App.—Austin 2000, no pet.). Once the movant specifies
    the elements on which there is no evidence, the burden shifts to the nonmovant to
    raise a fact issue on the challenged elements. TEX. R. CIV. P. 166a(i). A no-
    evidence summary judgment will be sustained when (1) there is a complete
    absence of evidence of a vital fact, (2) the court is barred by rules of law or of
    evidence from giving weight to the only evidence offered to prove a vital fact, (3)
    the evidence offered to prove a vital fact is no more than a scintilla, or (4) the
    5
    evidence conclusively establishes the opposite of a vital fact. King 
    Ranch, 118 S.W.3d at 751
    . We view the evidence in the light most favorable to the nonmovant,
    disregarding all contrary evidence and inferences. 
    Id. When a
    summary judgment motion is filed as a hybrid motion based upon
    both no-evidence and traditional grounds, we first review the trial court’s judgment
    under the no-evidence standard of review. See Ford Motor Co. v. Ridgway, 
    135 S.W.3d 598
    , 600 (Tex. 2004); All Am. Tel., Inc. v. USLD Commc’ns, Inc., 
    291 S.W.3d 518
    , 526 (Tex. App.—Fort Worth 2009, pet. denied). If the movant has
    filed a proper no-evidence motion for summary judgment and the non-movant has
    failed to produce more than a scintilla of evidence under the standards of Rule
    166a(i), there is no need to analyze whether the movant’s summary judgment proof
    satisfied the burden set forth for traditional summary judgment under Rule 166a(c).
    TEX. R. CIV. P. 166a(c), (i); E. Hill Marine, Inc. v. Rinker Boat Co., 
    229 S.W.3d 813
    , 816 (Tex. App.—Fort Worth 2007, pet. denied).
    COMPENSABLE INJURY
    The Texas Labor Code specifies when a heart attack is considered a
    compensable workers’ compensation injury:
    A heart attack is a compensable injury under this subtitle only if:
    (1)   the attack can be identified as:
    (A)    occurring at a definite time and place; and
    6
    (B) caused by a specific event occurring in the course
    and scope of the employee’s employment;
    (2) the preponderance of the medical evidence regarding the
    attack indicates that the employee’s work rather than the natural
    progression of a preexisting heart condition or disease was a
    substantial contributing factor of the attack; and
    (3) the attack was not triggered solely by emotional or
    mental stress factors, unless it was precipitated by a sudden
    stimulus.
    TEX. LAB. CODE ANN. § 408.008 (Vernon 2006).
    A.    Parties’ arguments
    Amerisure’s motion argued that there was no evidence of the following: (1)
    “the heart attack occurred at a definite time and place,” (2) “the heart attack was
    caused by a specific event occurring in the course and scope of the employee’s
    employment,” and (3) “the employee’s work rather than the natural progression of
    a preexisting heart condition or disease was a substantial contributing factor of the
    attack.” The principle focus of the parties’ arguments in the trial court and here
    has been this last factor, i.e., whether work was a “substantial contributing factor”
    of James’s heart attack.
    With regard to this element, Poplin’s summary-judgment response asserts
    that the “preponderance of the medical evidence regarding the attack indicates that
    the employee’s work rather than the natural progression of a preexisting heart
    condition or disease was a substantial contributing factor of the attack.” Her
    response further argued that no expert testimony was required on this element,
    7
    because the supreme court has held that no expert testimony is necessary to
    “decide the probabilities when the trier of the fact is given evidence of prompt
    onset of the [heart] attack following an occurrence competent to affect adversely a
    defective heart.” Ins. Co. of N. Am. v. Kneten, 
    440 S.W.2d 52
    , 54 (Tex. 1969). She
    also cites this Court’s opinion in Tex. Employers Indemnity Co v. Etie, 
    754 S.W.2d 806
    , 808 (Tex. App.—Houston [1st Dist.] 1988, no writ) for the proposition that,
    “[i]n a workers’ compensation case, expert testimony is generally not required to
    prove an issue of probability, if the trier of fact has been given sufficient evidence
    showing the prompt onset of symptoms following a specific event.”               Poplin
    acknowledges that the autopsy reflects that James had a preexisting heart
    condition, but she emphasizes that, after “first showing signs of heat exhaustion,
    prompt onset of the heart attack occurred and [he] was dead within the hour.”
    B.     Summary-judgment evidence
    Poplin’s summary-judgment response does not provide citations to any
    evidence, but attaches the following: (1) paramedic records from Houston Fire
    Department, (2) medical records from Christus St. John’s Hospital, (3) deposition
    excerpts of Albert Y. Chu, M.D. (assistant medical examiner), and (4) a letter from
    Ethan Podet, M.D. (cardiologist).2
    22
    In her summary-judgment response, Poplin objected to Amerisure’s reliance upon
    Chu’s testimony as an expert in the absence of a Robinson hearing. She also filed
    additional medical articles as summary-judgment evidence with her “Final
    8
    Chu’s deposition excerpts contain the following statements about causation:
    A.     Immediate cause of death is ineffectual pumping of the
    heart due to cardiac arrhythmia due to lechemia in his heart from
    blockages in his heart, coronary arteries, including the thrombus in his
    left anterior descending artery; but possibility also the blockages in his
    left circumflex artery, his right coronary artery, and the fact that he
    had an old myocardial interception and an enlarged heart from his
    long-standing hypertension.
    Q.    So you have no – in your last statement you gave, you
    had no opinion on what causes plaque to rupture?
    A.    In his specific case, no I don’t know what the inciting
    event or combination of events was.
    Q.     Well, what are some risk factors in plaque rupture, if you
    know? If you’ve got atherosclerosis, what are some risk factors that
    you want to – for example, you want to avoid so that you don’t have
    rupture of the plaque?
    A.      As I mentioned earlier, it’s a multi – it’s thought to be a
    multifactorial process. So there are certain intrinsic factors, such as
    the anatomy of the plaque itself, the anatomy of the heart, of the blood
    vessel itself, the person’s underlying medical condition. So certain
    health states lead one to be more likely to have plaque rupture.
    There are extrinsic factors, such as changes to blood pressure or
    blood flow; of course, whether or not the person is on drugs, such as
    cocaine; environmental factors, like heat; physical exertion. So there
    [are] many different potential triggers for plaque rupture. It could be
    a combination of those different things.
    Q.     Tell us about heat, how heat could apply in this situation
    in causing plaque rupture.
    Replication to Plaintiff’s Response on its Motion for Summary Judgment” to
    respond to some of the evidence Amerisure filed in support of its traditional
    motion for summary judgment. We need not address Poplin’s objections to
    Amerisure’s evidence or the evidence Poplin filed in response to Amerisure’s
    evidence, as both relate to Amerisure’s traditional motion for summary judgment,
    and we resolve this appeal on no-evidence grounds.
    9
    A.    Presumably, if someone’s hot and dehydrated, increasing
    the blood via – increasing the blood viscosity, that might cause more
    turbulence in the coronary arteries and lead to plaque rupture.
    Q.     In other words, the thickness of the blood would create
    additional stress on the walls of your arteries and possibility cause
    plaque to rupture, couldn’t it?
    A.      Possibly.
    Q.      Did you consider that in Mr. Poplin’s case?
    A.      I considered whether or not his death might have been
    heat related.
    Q.     Did you see from the Houston Fire Department records
    that he was administered saline IV’s on the way to the hospital
    because of fluid loss?
    A.      Yes.
    Q.    Okay, Did you consider that? Did you think that that
    could have been – or did you consider that as a possible cause of the
    plaque rupture in this case?
    A.      No.
    Q.      But it could have been a cause?
    A.     It could – it’s – I would say it could have been a risk
    factor for plaque rupture. However, as I mentioned earlier, I think
    he already had – his plaque had already ruptured by the time he
    went to work that day.
    Q.      Well, how did he – what makes you say that?
    A.     When I looked at the plaque microscopically, I saw
    evidence of inflammatory reaction to that thrombus.
    Q.      Where is that written here in your records?
    A.     It’s not in – it’s not in my report. I reviewed the slides
    this morning in preparing for this deposition.
    (emphasis added)
    10
    Podet’s letter stated that he had “reviewed the statements of Dr. Chu and
    Dana Andrews, the ER and EMT notes of 6-25-06, the autopsy of 6-27-06, and the
    medical records” and concluded the following with regard to causation:
    I agree with Dr. Albert Y. Chu, the assistant medical examiner,
    that the underlying cause of death was atherosclerotic cardiovascular
    disease and that the immediate cause was probably the LAD
    thrombus. The question has been raised as to whether Mr. Poplin’s
    strenuous work caused the heart attack, and cardiac arrest. Studies of
    physical exertion as a trigger of sudden death reveal that vigorous
    exertion multiplies the risk of sudden death 17-fold, compared to rest,
    but that the risk of sudden death for a specific episode of exertion is
    very low, at one excess death per 1.4 million episodes of exertion.
    (Albert CM. et al. Triggering of Sudden Death from Cardiac Causes
    By Vigorous Exertion. New Engl J Med 2000; 343:1355-61.)
    Given these statistical considerations, the nature of his death,
    and the chronic and acute cardiac disease found at autopsy, the cause
    of Mr. Poplin’s death was the natural progression of his underlying
    cardiac disease, rather than a specific episode of exertion during the
    course of his work.
    (emphasis added).
    C.    Analysis
    The question we must resolve is whether the evidence Poplin proffered is
    some “medical evidence . . . indicat[ing] that the employee’s work rather than the
    natural progression of a preexisting heart condition or disease was a substantial
    contributing factor of the attack.” TEX. LAB. CODE ANN. § 408.008(2). Poplin
    relies on the supreme court’s opinion in Kneten, a case holding that medical expert
    testimony was not necessary to establish an electric shock at work was a cause of a
    heart attack.
    11
    In the present case the fact finder had direct evidence of the
    occurrence on the job when the employee, while wet with sweat in the
    heat and effort of his work, was shocked throughout his body with an
    electrical current. The fact finder was told of the prompt onset of
    symptoms with the employee feeling bad within a few minutes and his
    distress progressing until he was in a critical state in the hospital
    within a few hours. The doctor testified that this distress was due to a
    heart attack and that the heart is still impaired. Further, the doctor
    testified that what happened on the job could precipitate a heart attack.
    With those facts given, it was not conjecture on the part of the jury to
    conclude that the occurrence on the job was probably a cause of the
    attack and resulting disability.
    Since the question is what precipitated this attack at this time, it
    requires no expert to decide the probabilities when the trier of fact is
    given evidence of prompt onset of the attack following an occurrence
    competent to affect adversely a defective heart. As in all of those
    cases where a back injury promptly follows a lifting strain, or a
    ruptured blood vessel or heart attack promptly follows exertion,
    though there is not definite proof of the mechanical process by which
    the physical structure of the body is damaged, under the
    circumstances it is reasonable to believe that what the employee did
    on the job precipitated physical failure. The courts have often allowed
    this finding and permitted recovery under the Texas Workmen’s
    Compensation 
    Law. 440 S.W.2d at 53
    –54 (citations omitted).
    According to Poplin, Chu’s statement that dehydration “might cause more
    turbulence in the coronary arteries and lead to plaque rupture” combined with
    Kneten’s recognition that a fact finder can—without an expert—conclude that an
    occurrence preceding a heart attack “was probably a cause of the attack,” 
    id., satisfies section
    408.008(2)’s requirement that “the preponderance of the medical
    evidence regarding the attack indicates that the employee’s work rather than the
    12
    natural progression of a preexisting heart condition or disease was a substantial
    contributing factor of the attack.” We disagree.
    Preliminarily, we note that Kneten—decided in 1969—predates the current,
    applicable statutory standard for determining when a heart attack is a compensable
    injury under the worker’s compensation laws.        As the San Antonio Court of
    Appeals has explained, “[p]rior to 1989, there was no specific statute dealing with
    the compensability of heart attacks.” Transcon. Ins. Co. v. Smith, 
    135 S.W.3d 831
    ,
    836 (Tex. App.—San Antonio 2004, no pet.). “The case law that developed before
    1989 focused on the issue of whether an employee’s strenuous work activities
    caused the heart attack even though the employee had a pre-existing heart
    condition.” 
    Id. “[C]ourts only
    required that the job-related strain be at least a
    contributing cause of the heart attack.”     
    Id. “Therefore, if
    the work incited,
    accelerated, or aggravated the claimant’s underlying heart condition, it was a
    sufficient cause of the resulting heart attack for the purposes of recovering
    workers’ compensation benefits.” 
    Id. In contrast,
    the current statute requires “the
    employee’s work rather than the natural progression of a preexisting heart
    condition or disease [be] a substantial contributing factor of the attack.” TEX.
    LABOR CODE ANN. § 408.008(2) (emphasis added).
    “Additionally, before 1989, medical testimony was not required on whether
    an occurrence caused a heart attack.” Transcontinental Ins. 
    Co., 135 S.W.2d at 13
    836. Now the Labor Code requires us look to the “the preponderance of the
    medical evidence regarding the attack” in determining whether the employee’s
    work was a substantial contributing factor. TEX. LABOR CODE ANN. § 408.008(2)
    (emphasis added).
    For these same reasons, the Fourteenth Court of Appeals has questioned
    Kneten’s continued viability in the worker’s compensation context. See Choice v.
    Gibbs, 
    222 S.W.3d 832
    , 837 n.5 (Tex. App.—Houston [14th Dist.] 2007, no pet.)
    (“In workers’ compensation cases, a question might arise regarding the extent to
    which the Kneten opinion has survived the enactment of this statute.”).
    By the plain language of the statute, to survive Amerisure’s no-evidence
    motion for summary judgment, Poplin was required to proffer in response
    “medical evidence regarding the attack indicat[ing] that the employee’s work
    rather than the natural progression of a pre-existing heart condition or disease was
    a substantial contributing factor.” TEX. LAB. CODE § 408.008(2). We have found
    this standard satisfied by expert physician testimony that (1) “higher-than-normal
    activity can increase the risk for a heart attack,” (2) the day before an employee’s
    heart attack, he engaged in physical activity that was “significantly higher” than
    usual, (4) “from a cardiac perspective, [the employee] had been stable up until” the
    strenuous work activity, (4) the employee suffered plaque rupture, causing his
    heart attack and death, and (5) “it was [the employee’s] activity [at work] that
    14
    caused the rupture.” New Hampshire Ins. Co. v. Allison, No. 01-12-00505-CV, __
    S.W.3d __, 
    2013 WL 3947822
    , at *4, 9 (Tex. App.—Houston [1st Dist.] Aug. 1,
    2013, no pet. h.).
    Poplin’s evidence falls short of what we have previously held sufficient
    under section 408.008(2). Her summary-judgment evidence here consists of: (1)
    medical records reflecting that James suffered dehydration at work, (2) medical
    records reflecting that shortly after suffering dehydration, James had a heart attack,
    (3) Chu’s deposition testimony that heat and dehydration can “possibly” cause
    plaque to rupture, (4) Chu’s deposition testimony that he did not consider heat or
    fluid loss to be a possible cause of James’s plaque rupture because “his plaque had
    already ruptured by the time he went to work that day,” and (5) Podet’s opinion
    that “the cause of [James’s] death was the natural progression of his underlying
    cardiac disease, rather than a specific episode of exertion during the course of his
    work.” Because Poplin did not proffer evidence that James’s “work rather than the
    natural progression of a pre-existing heart condition or disease was a substantial
    contributing factor” his heart attack, the trial court did not err in granting
    Amerisure’s no-evidence motion for summary judgment.
    15
    CONCLUSION
    We affirm the trial court’s judgment.
    Sherry Radack
    Chief Justice
    Panel consists of Chief Justice Radack and Justices Bland and Huddle.
    16